SIU

BIOTECHNOLOGY - MICR 421


Chapter 15. Microbial Insecticides

-Bacillus thuringiensis

-Baculovirus


Insecticides

Major uses: Crop protection & control of disease vectors (e.g. mosquitoes)

Problems asssociated with chemical insecticides

Degrade slowly: persist in the environment (15-20 years for DDT)

Pollution of surface and groundwater

Bioaccumulation: concentration in animal tissues is magnified through the food chain

algae, bacteria ---> daphnia ---> fish ---> eagles, humans etc.

Nonselective: toxic to nontarget organisms (wildlife, beneficial insects, farm animals, humans)

Development of resistance by target organisms

Requires application of more insecticide and introduction of new insecticides

 

Microbial insecticides have fewer problems

Ex. Toxins produced by bacteria (Bacillus thuringiensis)

Pathogens that infect insects (Bacillus popilliae, Baculovirus)


Bacillus thuringiensis as an insecticide

Gram + spore former, produces a protein that kills insects

See Figs. 15.1 and 15.3

Several subspecies are toxic to different insect orders

See Table 15.1

Subsp. toxicities

kurstaki ---> lepidopteran larvae (moths, butterflies)

israelensis ---> dipteran larvae (mosquitoes)

tenebrionis ---> coleopterans (potato beetle, boll weevil)

Disadvantages

Applied by spraying plant surfaces and must be ingested by insect

Boring, sucking and root eating insects not affected

Kill insect only at a particular stage of insect's life cycle

Timing of application is crucial

Specificity

Use of just one subsp. will not kill multiple crop pests

Expensive

Ex. kurstaki costs 1.5-3 X more than chemical insecticides

Rapidly degraded by sunlight

May not persist long enough for control of insect pest

(+) However, this prevents development of resistance


Cloning cry Genes from Bacillus thuringiensis subsp. kurstaki

See Fig. 15.4

Cloning procedures

1. Plasmids were separated from chromosomal DNA by centrifugation through a gradient of cesium chloride

2. Small plasmids were discarded because they were not big enough to code for a protein with a molecular weight of 130,000 (130 k-Da).

3. Medium and large plasmids were separated from each other by centrifugation through a sucrose gradient.

4. The medium and large plasmids were partially digested with Sau 3AI and ligated into the Bam HI restriction site of the cloning vector pBR322.

Why was it possible to ligate DNA cut with the two different restriction enzymes?

5. The DNA was introduced into E. coli by transformation to create a DNA library.

6. The library was screened for the expressed toxin using an immunoassay.

The immunoassay used an antibody that bound to the toxin protein.

The antibody was radioactively labeled by binding protein A containing a radioactive isotope of iodine, 125I.

Protein A is produced by Staphylococcus aureus and binds to the constant domain of antibodies.

Screening consisted of exposing a membrane colony lift to the radioactive antibody which was detected by autoradiography


Genetic Engineering B. thuringiensis and cry Genes

1. Express toxin gene continuously during vegetative growth

See Fig. 15.5

Most cry genes expressed only during sporulation phase of growth

i.e. at the end of the growth cycle

Sporulation specific promoters control expression

Place cry gene under control of promoter of a gene that is constitutively expressed during vegetative growth

Ex. Tetr gene or cryIIIA gene

Higher yields of toxin would be produced in less time

 

2. Broaden spectrum of toxicity against more insect species

i) Transform B. thuringiensis strains with several cloned cry genes that encode proteins that kill different insect species

See Table 15.3

 

3. Increase availability of toxin to insects by introducing cry genes into other species of bacteria

i) Mosquito control

Heavy parasporal crystals sink below feeding zone of mosquitoe larvae

Transfer cry genes to aquatic microorganisms that inhabit feeding zone and are food source for mosquito larvae

Cyanobacteria (photosyntheitc), Caulobacter crescentus, Asticcacaulis excentricus

Ex. Asticcacaulis excentricus

Dwells near surface and doesn't sink

Grows on a cheap medium

Low protease activity protects protoxin from inactivation

Adapted to high levels of UV light present near water surface

 

ii) Control of insects that feed on plant roots

Spraying crops w/ B.t. doesn't kill insects that feed on roots

Pseudomonas fluorescens inhabits rhizosphere

Protect roots by transferring cry genes to this organism

Inoculate seeds w/ recombinant organism before planting

iii) Control of sucking and boring insects

Ingest very little protoxin during feedong

Clavibacter xyli inhabits xylem of grasses

Transfer of cry gene to C. xyli protects corn from dmage by European corn borer


Baculoviruses

See Table 15.5

Ex. pine saw fly, cotton bollworm, gypsy moth, cabbage looper

Advantages/disadvantages

+/ - Limited host ranges

Want to controll multiple pests but not to affect beneficial insects

+ Resistance of insect to virus rarely develops

- Slow to kill

May still cause crop damage before dying

- High production costs

Viruses must be produced in cultured insect cells (or whole insects)

 

Genetic Engineering of Baculoviruses

1. Introduce genes that disrupt levels of hormones controlling stages of insect life cycle.

Ex. some insect hormones are esters: introduce esterase gene.

Esterase

R-C=O(O-CH3) + H2O ------------------> R-COOH + CH3OH
(juvenile hormone) ...............................................INACTIVE

Treated insect larvae stopped growing and feeding on plant

-Disadvantage: hormone's influence occurs only at specific stage of life cycle, making the timing of application to crops critical for effectiveness

 

2. Introduce genes that increase viral pathogenicity.

Ex. Scorpion gene that encodes an insect-specific neurotoxin

Insects infected with the engineered baculovirus caused less plant damage than those infected with the wild-type virus

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Last updated: November 10, 2003 /jdh

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